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Pustular psoriasis: Precipitating factors: Pustulation precipitated by: Irritant effect of topical therapyWithdrawal of topical or systemic steroids Clinical features: Pustulation can be: Localized: as seen in palmoplantar psoriasisGeneralized: as seen in von Zumbusch’s pustular psoriasis which is:A serious condition accompanied by constitutional symptoms (high fever, chills, tachypnea). Characterized by generalized fiery red erythema followed by appearance of tiny waves of superficial pustules (often can be easily wiped off), which become confluent to form circinate lesions and lakes of pus. Appearance of new pustules as the old ones are crusting. Enrol to Pathology revision series and get the entire video till NEET + the GT's - Subscribe here today - 2 days enroll before early bird ends.. Finishline Revision starts today ! Do not miss it !!

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Oral lichen planus (OLP) is a chronic inflammatory disease that causes bilateral white striations, papules, or plaques on the buccal mucosa, tongue, and gingivae. Erythema, erosions, and blisters may or may not be present Patterns of oral lichen planus. The reticular pattern (see right) is commonly found on the cheeks as lacy web-like, white threads that are slightly raised. These lines are sometimes referred to as Wickham’s Striae. The name lichen comes from a plant that is often seen growing on rocks with its mossy, web-like appearance.The erosive (atrophic) pattern can affect any mucosal surface, including the cheeks, tongue, and gums Revision Finishline starts from today. Have daily tests (topic wise) and GT and get all the pathology videos.  Enrol to Pathology revision series and get the entire video till NEET + the GT's - Subscribe here today - 2 days enroll before early bird ends. ! 

Finishline

Dear Students,  So here we again start after a break. We shall have schedule ways from now on. I want you to spend 1 to 2 hours (Maximum for daily topics). Read in the morning - You will have the test by evening as usual #8to9ispathotime  We will be taking baby steps towards our mammoth NEET and lets destroy it. Everyday we will be have a small pathology topic combined with a part of another subject. I want this exercise to be done in addition to your existing schedule. Since you are revising it again it will not take more than 2 hours to read the topic for everyday.  Next week schedule will be updated soon. !! This will be available for all revision series users - Enrol to Pathology revision series and get the entire video till NEET + the GT's - Subscribe here today - 2 days enroll before early bird ends. ! 

Power Combo - Ophthalmology and Pathology

Dear Students, Thank you for the response for the 2 days live session at Delhi. March 18 - Ophthalmology (Dr. Rajarathna)March 19 -  Pathology (Dr. Ranjith) Mark your dates, finish reading Ophthalmology and Pathology with us ! What's ap for Registration - http://wa.me/919751295255 (Less than 10 seats left for the 500 rs discount). Hurry to book your slots. !

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Vitiligo - treatment options available: No medical treatment (use cosmetics to add lost color): Cosmetic options include makeup, a self-tanner and skin dye.Medicine applied to the skin: Steroids, tacrolimus, pimecrolimusLight treatment:PUVA light therapy.SurgeryUnconventional treatment – Gingo biloba, vitaminsDepigmentationEnrol to Pathology revision series and get the entire video till NEET + the GT's - Subscribe here today - 2 days enroll before early bird ends. ! Post Feb 11 -  MRP - Rs. 1399/- 

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Dermatophytes: Causative agent of Ringworm or Tinea or Dermatophytoses .Not involve living Tissues.It has 3 genera – Trichophyton, Microsporum , Epidermophyton.In lesion, it form hyphae and arthospores. In culture it form septate hyphae and asexual spores (micro and macroconidia) with powdery and pigmented colonies.They are differentiated mainly by nature of macrocondia. Clinical features Local inflammation is due to irritation by fungal products and hypersensitivity reaction.Transmission occurs from infected to uninfected person often by brushes, combs and towels. Clinically ringworm is classified depending on the site involved eg. Tinea capitis infect scalp and hair.MC species infecting human being – T. rubrum Kerion The fungal infection of scalp caused by microsporum or trichophyton species not by epidermophyton. Inflammatory boggy swelling covering small or large areas of scalp in which hair are loose and fallout or can be easily epilated.Commonly caused by zoophilic dermatophytes like T. mentagrophytes and T. verrucosum.Follicular scarring and partial alopecia is common after severe kerion.Pathology Revision + GT - Rs. 999/- You will get access to all till NEET - You can enrol to Subscribe here today 

Forensic Images Uploaded

A few of Handpicked images in Forensic Medicine uploaded. It will be available in the Image Bank for revision users. ! We as a team are all geared up for the first GT....!The aim is simple  - Attend all 300 questions.! Lets do it and analyse the errors. Enrol to the course which suits you. Soft copy notes will be uploaded in two weeks time !Complete Pathology course - Discounted Price -   Subscribe here todayPathology course with Practicals -  Discounted Price -   Subscribe here todayPathology Revision + GT - Rs. 999/- You will get access to all till NEET - You can enrol to Subscribe here today 

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Pharmacokinetics of Gliptins Sitagliptin is well absorbed orally, is little metabolized and is largely excreted unchanged in urine with a t½ averaging 12 hours. Dose reduction is needed in renal impairment, but not in liver disease. Vildaglitptin: The major route of elimination of vildagliptin is by hepatic metabolism; only 20–25% is excreted unchanged in urine. Dose reduction is needed in moderately severe liver and kidney disease. Linagliptin lowers HbA 1c by 0.4–0.6% when added to metformin, sulfonylurea, or pioglitazone. The dosage is 5 mg daily and since it is primarily excreted via the bile, no dosage adjustment is needed in renal failure. Saxagliptin is given orally as 2.5–5 mg daily. The drug reaches maximal concentrations within 2 hours (4 hours for its active metabolite). It is minimally protein bound and undergoes hepatic metabolism by CYP3A4/5. The major metabolite is active, and excretion is by both renal and hepatic pathways. The terminal plasma half-life is 2.5 hours for saxagliptin and 3.1 hours for its active metabolite. Dosage adjustment is recommended for individuals with renal impairment Pathology Revision + GT - Rs. 999/- You will get access to all till NEET - You can enrol to Subscribe here today

Bone Tumour Pathology

Listen to the Pathogenesis of Exostosis. Subscribe to the Channel for more videos and live sessionsEnrol to lectures to watch the complete videos !Complete Pathology course - Discounted Price -   Subscribe here todayPathology course with Practicals -  Discounted Price -   Subscribe here todayPathology Revision + GT - Rs. 999/- You will get access to all till NEET - You can enrol to Subscribe here today 

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Contributory negligence: A doctrine of common law that if a person was injured in part due to his/her own negligence (his/her negligence "contributed" to the accident), the injured party would not be entitled to collect any damages (money) from another party who supposedly caused the accident Last chance clear doctrine: The rule of last clear chance operates when the plaintiff negligently enters into an area of danger from which the person cannot extricatehimself or herself. The defendant has the final opportunity to prevent the harm that the plaintiff otherwise will suffer. The doctrine wasformulated to relieve the severity of the application of the contributory negligence rule against the plaintiff, which completely bars anyrecovery if the person was at all negligent. There are four types: Helpless Plaintiffs Inattentive Plaintiffs Observant Defendant Inattentive DefendantEnrol to Pathology revision series and get the entire video till NEET + the GT's - Subscribe here today - 3 days enroll before early bird ends. ! Post Feb 11 -  MRP - Rs. 1399/- 

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